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Metabolic remodeling in pulmonary artery smooth muscle cells (PASMCs) and pulmonary artery endothelial cells (PAECs), including enhanced glycolysis and mitochondrial dysfunction, provides the energy and material basis for abnormal proliferation of vascular cells (Xu et al., 2024

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OxLDL loading significantly depleted cytosolic and mitochondrial anisotropy in a time-dependent manner (Fig 4D), suggesting that oxLDL loading increased GSR-mediated NADPH consumption in both compartments